L66.81

Clinical Causes: Genetic predisposition: Family history can increase risk. Hair care practices: Use of harsh chemical treatments, relaxers, or heat styling tools may contribute. Inflammation: Chronic scalp inflammation damages hair follicles over time. Autoimmune factors: Some evidence suggests immune system components may play a role. Environmental factors: External irritants and environmental damage may influence disease progression.

Key Symptoms: Gradual, painless hair loss, often starting at the crown of the scalp. Scaling, redness, or inflammation on the scalp surface. Formation of scar tissue replacing hair follicles. Changes in scalp texture, such as become firm or ridged. In some cases, hair loss may be asymptomatic initially, leading to delayed diagnosis.

Diagnosis Path: Clinical examination of the scalp, assessing the pattern of hair loss and signs of scarring. Dermatoscopic evaluation to examine scalp and hair follicles more closely. Scalp biopsy: A small tissue sample taken for microscopic analysis to confirm scarring and follicle destruction. Medical history review, including hair care practices and family history. Differential diagnosis to distinguish from other types of alopecia, such as male or female pattern baldness.

Treatment Protocols: Anti-inflammatory medications: Corticosteroid creams or injections to reduce scalp inflammation. Hair growth stimulants: Topical minoxidil may be recommended to encourage hair regrowth, though effectiveness varies. Avoidance of harsh hair treatments: Limiting chemical relaxers, heat styling, and tight hairstyles. Hair transplant surgery: In some cases, transplantation of healthy hair follicles can be considered. Monitoring and follow-up: Regular medical reviews to assess disease progression and adjust treatment strategies.